There's nothing particularly unbelievable about that. There are 4 possible base pairs, the human genome contains 3 billion base pairs, 98% of it noncoding (and noncoding doesn't mean non-functional, but 98% of the genome isn't regulatory either). Every single 3-base pair combination codes for an amino acid, an initiation site or a stopping site. The odds that there exist short strings of codons in there that correspond to a functional protein and are just lacking an ORF isn't low at all. If those had been very long genes it would be another story but in fact they're very very short.
I really don't see how we can go further with this, can't we go back to the other points we were talking about before discussing this article took over the whole conversation ?
(although I would still like to find out more about those gibbons with chronic lymphocytic leukemia, I still haven't been able to find anything on that, maybe you could give me google search terms with which to find out more if nothing else ?)
I'm sorry, if you cannot at least admit to the possibility of those four apes independently having that disabler, then I see you are far too stubborn to be able to discuss issues with. Why discuss issues if there are going to be no conclusions?
You are referring to 4 possible base pairs , I am referring to entire genes in humans involving thousands of base-pairs each. You say this gene was never coding, and humans evolved, and this became a coding region. I don't think you quite get the lack of randomness in genes. Its like a computer code, a precise set of information that produces precise functions in the human body through the production of proteins. You seem to brush over the statistical impossibility of this spontaneously occurring across a unique combination of thousands of unique base-pairs. Our previous discussion involved the possibility of duplicating and changing a gene, now we are talking about novel functional genes in a non-coding format waiting to be activated. Maybe your wording implies that this is all natural and possible, but only a complete idiot would think that an entire non-coding region could suddenly become coding and contribute non-damaging proteins in a functional manner (eg producing specific antibodies) . Production of proteins has to be done in a very precise balanced manner, it cannot occur across a few novel unique genes spontaneously and in full functional form.